Ganong's Review of Medical Physiology 25th EditionThe leading text on human physiology for more than four decades For more than four decades, Ganong’s Review of Medical Physiology has been helping those in the medical field understand human and mammalian physiology. Applauded for its interesting and engagingly written style, Ganong’s concisely covers every important topic without sacrificing depth or readability and delivers more detailed, high-yield information per page than any other similar text or review. Thoroughly updated to reflect the latest research and developments in important areas. Ganong’s Review of Medical Physiology incorporates examples from clinical medicine to illustrate important physiologic concepts.
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Page 24
... glucose CH2 OH P O 1:4α linkage O Glucose- 1-phosphate Glucose- 6-phosphate bloodstream. The principal determinants are therefore the dietary intake; the rate of entry into the cells of muscle, adi- pose tissue, and other organs; and ...
... glucose CH2 OH P O 1:4α linkage O Glucose- 1-phosphate Glucose- 6-phosphate bloodstream. The principal determinants are therefore the dietary intake; the rate of entry into the cells of muscle, adi- pose tissue, and other organs; and ...
Page 446
... glucose Blood lactate Muscle glycogen % Time after injection of epinephrine (h) FIGURE 24–15 Effect of epinephrine on tissue glycogen, plasma glucose, and blood lactate levels in fed rats. (Reproduced with permission from Ruch TC ...
... glucose Blood lactate Muscle glycogen % Time after injection of epinephrine (h) FIGURE 24–15 Effect of epinephrine on tissue glycogen, plasma glucose, and blood lactate levels in fed rats. (Reproduced with permission from Ruch TC ...
Page 478
... glucose and galactose is driven secondarily by the low intracellular sodium concentration established by the basolateral Na+, K+ ATPase (not shown). SGLT-1, sodium-glucose cotransporter-1. Trypsin FIGURE 26–3 Mechanism to avoid ...
... glucose and galactose is driven secondarily by the low intracellular sodium concentration established by the basolateral Na+, K+ ATPase (not shown). SGLT-1, sodium-glucose cotransporter-1. Trypsin FIGURE 26–3 Mechanism to avoid ...
Contents
Cellular Molecular Basis for Medical Physiology | 1 |
Central Peripheral Neurophysiology | 157 |
Endocrine Reproductive Physiology | 297 |
Copyright | |
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abnormal acid action activity addition alveoli amino acids amount appears arterial associated axons binding blood body bone brain Ca2+ called capillaries cardiac cause cells changes channels Chapter circulation Clinical Box complex concentration conduction contain contraction cortex cytoplasm decrease described direct disease drugs effects enter enzyme example factor fibers Figure flow fluid function gene glands glucose growth heart hormone humans hypothyroidism important increase individuals inhibition initial insulin intestine involved leads liver located loss lungs mechanisms membrane molecules motor move movement muscle myosin nerve neurons normal nucleus occurs organs pain pathways patients permission Physiology placenta plasma potential pressure produced protein receptors reduced reflex regulation relatively release response result rise secretion sensory smooth spinal stimulation structure substances Table terminal thyroid tion tissues tract transport treatment types various vessels volume