Ganong's Review of Medical Physiology 25th EditionThe leading text on human physiology for more than four decades For more than four decades, Ganong’s Review of Medical Physiology has been helping those in the medical field understand human and mammalian physiology. Applauded for its interesting and engagingly written style, Ganong’s concisely covers every important topic without sacrificing depth or readability and delivers more detailed, high-yield information per page than any other similar text or review. Thoroughly updated to reflect the latest research and developments in important areas. Ganong’s Review of Medical Physiology incorporates examples from clinical medicine to illustrate important physiologic concepts.
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Page 67
... tissues. Likewise, the immune system clears the body's own cells that have become senescent or abnormal, such as cancer cells. Finally, normal host tissues occasionally become the subject of inappropriate immune attack, such as in ...
... tissues. Likewise, the immune system clears the body's own cells that have become senescent or abnormal, such as cancer cells. Finally, normal host tissues occasionally become the subject of inappropriate immune attack, such as in ...
Page 327
... tissues Diverse tissues Level regulated by Glucose Growth hormone after birth, nutritional status Unknown Plasma levels 0.3–2 ng/mL 10–700 ng/mL; peaks at puberty 300–800 ng/mL Plasma- binding proteins No Yes Yes Major physiologic role ...
... tissues Diverse tissues Level regulated by Glucose Growth hormone after birth, nutritional status Unknown Plasma levels 0.3–2 ng/mL 10–700 ng/mL; peaks at puberty 300–800 ng/mL Plasma- binding proteins No Yes Yes Major physiologic role ...
Page 597
... tissue prekallikrein into tissue kallikrein is not known. Endothelial cell surface Tissues Plasma prekallikrein Tissue prekallikrein ? C1INH Tissue kallikrein Factor XII Factor XIIa LMW kininogen Kallidin HMW kininogen Bradykinin Plasma ...
... tissue prekallikrein into tissue kallikrein is not known. Endothelial cell surface Tissues Plasma prekallikrein Tissue prekallikrein ? C1INH Tissue kallikrein Factor XII Factor XIIa LMW kininogen Kallidin HMW kininogen Bradykinin Plasma ...
Contents
Cellular Molecular Basis for Medical Physiology | 1 |
Central Peripheral Neurophysiology | 157 |
Endocrine Reproductive Physiology | 297 |
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abnormal acid action activity addition alveoli amino acids amount appears arterial associated axons bile binding blood blood flow body bone brain Ca2+ called capillaries cardiac cause cells changes channels Chapter circulation Clinical Box complex concentration conduction contain contraction cortex cytoplasm decrease described direct disease drugs effects enter enzyme example factor fibers Figure fluid function gene glands glucose growth heart hormone humans hypothyroidism important increase individuals inhibition initial insulin intestine involved leads liver located loss lungs mechanisms membrane molecules motor move movement muscle myosin nerve neurons normal nucleus occurs organs pain pathways patients permission Physiology placenta plasma potential pressure produced protein receptors reduced reflex regulation relatively release response result rise secretion sensory smooth spinal stimulation structure substances Table terminal thyroid tion tissues transport treatment types various vessels volume